Ever heard of a thighbone break that looks different from a regular fracture? That’s an atypical femur fracture, and it often shows up in people taking certain bone‑strengthening drugs for years.
If you’ve been on bisphosphonates (like alendronate or risedronate) for over five years, your risk jumps. The medication helps prevent osteoporosis but can also make the outer layer of the femur too rigid. Older adults, especially post‑menopausal women, see this most often. Even people without obvious bone loss can develop a tiny crack on the side of the thighbone that slowly worsens.
Other red flags include long‑term steroid use, low calcium intake, and having had a previous femur fracture. A sudden sharp pain in the upper thigh or groin while walking, even without falling, should set off an alarm.
The first step is getting an X‑ray or MRI to spot that characteristic thin line on the bone. Doctors may order a CT scan if the picture isn’t clear. Once confirmed, treatment usually starts with stopping the bisphosphonate and switching to another osteoporosis drug, like denosumab.
Most small cracks can heal with protected weight‑bearing – think crutches or a walking boot for six weeks. Larger breaks often need surgical fixation using a metal rod inside the femur. Physical therapy kicks in early to keep the muscles strong while the bone repairs.
Prevention is easier than cure. Talk to your doctor about taking drug holidays after five years of bisphosphonate use, getting enough vitamin D and calcium, and staying active with low‑impact exercises like swimming or cycling.
Bottom line: an atypical femur fracture isn’t a random accident; it’s usually a signal that something in your bone health plan needs tweaking. Spotting the pain early, getting proper imaging, and adjusting medication can keep you on your feet without surgery.